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[7] This dual route is the mechanistic feature that most clearly separates survodutide from GLP-1 mono-agonists in the liver-disease setting, where reducing hepatic fat is the entry point to slowing inflammation and, eventually, fibrosis

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Moreover, if dexamethasone is not available, alternative glucocorticoids such as prednisone (40 mg), methylprednisolone (32 mg), or hydrocortisone (160 mg) can be used

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