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After 1 year of UDCA, 110 patients (81%) achieved an UDCA response

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When TLR4 is activated by bacterial LPS or by molecules from injured cells, it triggers a cascade of events that causes the cell to produce inflammatory cytokines.[ref] LDN is thought to block the activation of TLR4, thus reducing the release of inflammatory cytokines such as TNF-alpha, IL-6, and IL-12.[ref] LDN is one of the few TLR4 antagonists that can cross the blood-brain barrier.[ref] An animal study of multiple sclerosis found that naltrexone prevents the learning and memory issues associated with neuroinflammation

6.2 Diet and MCT Although the understanding of the pathophysiology of lcFAODs has increased significantly in the past decades, treatment options are still limited [144]