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Most importantly, this animal model has predictive value as it demonstrates biomarkers of abnormal mitochondrial fatty-acid metabolism (that is, acyl-carnitine elevations) that could be used as routine biomarkers if found in children with ASD

CPT1A P479L mutation decreases glucose concentrations and changes the lipoprotein profile Following the evaluation of pathological motor and non-motor behavior, we hypothesized that downregulation of CPT1 and specifically CPT1A would result in a shift toward glucose metabolism

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