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To survive the deleterious effects of the oxidative stress, tumor cells have adapted to utilizing the cellular biochemical and molecular anti-oxidant machinery, and thus have high dependency on cellular anti-oxidant molecules [1]

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The ensuing lactylation modifications of VE-cadherin and VEGFR2 are closely linked to LDHA levels, which in turn exacerbate the progression of GBM [154]

Tumour necrosis factor-alpha receptor 1 and 2 polymorphisms in inflammatory bowel disease and their association with response to infliximab