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Comprehensive analysis supports the fundamental difference between senescence and eryptosis, evidenced primarily by much faster clearance of eryptotic cells compared to senescent erythrocytes [78]

Because of the lethal potential of cyanide toxicity, most organisms have developed effective detoxification mechanisms that classically require sulfur donors, primarily sulfur-containing amino acids, to convert CN into thiocyanate (SCN), a putatively less toxic molecule 9

Previous studies suggested that ferroptosis is executed by peroxidizing PUFA-chains-containing phospholipids (PL), especially phosphatidylethanolamines (PE) and phosphatidylcholines (PC) [44], while the lysophospholipids (LP) increase upon ferroptosis inducer treatments as the product of enzymatic cleavage of the oxidized PUFA tail [45, 46]

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