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Current evidence suggests: its primary target is NNMT metabolism rather than endocrine hormone receptors

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Consistent with its role in LD metabolism, confocal microscopy showed that HSL prominently co-localized with LDs in M2 macrophages, a pattern that was less evident in M1 cells (Fig

Since HSL is the enzyme that is said to support fat cell breakdown by breaking down stored triglycerides into free fatty acids and glycerol, its activation may lead to a reduction in fat cell size